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Latest MSG study in rural China shows that MSG contributes to obesity in humans, regardless of caloric intake and activity.

Ahmad, K. and K. Jahan.  Studies on the Preventative and Curative Action of Ascorbic Acid on the Neurological Toxicity of Monosodium Glutamate. Institute of Nutritional and Food Science - University of Dhaka.

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Canfield, P.R. et al. 1992. Aspartame Exacerbates EEG Spike-wave Discharge in Children with Generalized Absence Epilepsy: A Double-blind Controlled Study. Neurology 42: 1000-1003.

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Choi, Dennis W. 1998. Manipulating Glutamatergic Neurotransmission for Therapeutic Gains: The Example of Brain Ischemia. Speaker, NIH National Meeting. The Glutamate Cascade: Common Pathways of Central Nervous System Disease States. May3-5, 1998.

Conner-Kerr, Teresa Ann. 1992. A Possible Mechanism of Excitotoxicity: Autoregulation of Glutamate Release from Hippocampal Mossy Fiber Terminals. Ph.D. dissertation, East Carolina University. Dissertation Abstracts Online. 53(11B): 5571.

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Dawson, R., Jr. et al. 1989. Age and Dose Dependent Effects of Neonatal Monosodium Glutamate (MSG) Administration To Female Rats. Neurotoxicol.  Teratol. 11 (4) 331-337.

Dawson, R., Jr. et al. 1989. A Pharmacological Anaysis of Food Intake Regulation in Rats Treated Neonatally with Monosodium L-Glutamate (MSG). Pharmacol.  Biochem. Behav. 32 (2): 391-398.

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De Groot, A. P. et al. 1988. Induction of Hyperplasia in the Bladder Epithelium of Rats by a Dietary Excess of Acid or Base: Implications for Toxicity/Carcinogenicity Testing. Food and Chemical Toxicology 26 (5): 425-434.

De Schutter, Erik, 1997. A New Functional Role for Cerebellar Long Term Depression. Progress in Brain Research 114:529-542.

Dolnikoff, M. S. et al. 1988. Neonatal Treatment with Monosodium Glutamate Increases Plasma Corticosterone in Rat.   Neuroendocrinology 48 (6): 645-649.

Dolnikoff, M. S. et al. 2001.  Decreased Lipolysis and Enhanced Glycerol and Glucose Utilization by Adipose Tissue Prior to Development of Obesity in Monosodium Glutamate (MSG) treated Rats.  Int. Journal of Obesity-Related Metabolic Disorders.

During, M.J. and Spencer, D.D. 1993.  Extracellular Hippocampal Glutamate and Spontaneous Seizure in the Conscious Human Brain Lancet 341:1607-0610.

Dzogas, Zoran. 1996.  In Vivo Activity of Canine Expiratory Bulbospinal Neurons is Differentially Affected by two Non-N-Methyl-D-Aspartate Receptor Antagonists. Croatian Medical Journal  37(1).

Eck, H. P. et al. 1990. Partial Recovery of Lymphocyte Activity in Patients with Colorectal Carcinoma After Curative Surgical Treatment and Return of Plasma Glutamate Concentrations to Normal Levels. Journal of Cancer Research and Clinical Oncology 116(6): 648-50.

Erdo, S. L. and M. Schaefer. 1991. Memantine is Highly Potent in Protecting Cortical Cultures Against Excitotoxic Cell Death Evoked by Glutamate and N Methyl-D-Aspartate Eur. Journal of Pharmacology 198 (2-3): 215-218.

Favaron, M. et al. 1990. Down-regulation of Protein Kinase C Protects Cerebellar Granule Neurons in Primary Culture from Glutamate-Induced Neuronal Death. Proceedings of the National Academy of Sciences of the United States of America 87 (5): 1983-1987.

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Fernstrom, J.D. 2000.  Pituitary Hormone Secretion in Normal Male Humans: Acute Responses to a Large, Oral Dose of Monosodium Glutamate. Journal of Nutrition 130 (Suppl 4s):1053-7.

Fisher, R. S. 1991. Glutamate and Epilepsy. Frontiers of Clinical Neuroscience 11: 131-146.

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Frandsen, A. and Arne Schousboe. 1991. Dantrolene Prevents Glutamate Cytotoxicity and Calcium Release From Intracellular Stores in Cultured Cerebral Corical Neurons. Journal of Neurochemistry 56 (3): 1075-8.

Frankel, Arthur et al. 1990. Role of Arginine 180 and Glutamic Acid 177 of Ricin Toxin A Chain in Enzymic Inactivation of Ribosomes. Molecular Cell Biology 10 (12) 6257-63.

Freese, A. et al. 1990. Characterization and Mechanism of Glutamate Neurotoxicity in Primary Striatal Cultures. Brain Research 521 (1-2): 254-264.

Fundytus, M.E., 2001. Glutamate Receptors and Nociception: Implications for the Drug Treatment of Pain. CNS Drugs 2001 Jan: 15(1): 29-58.

Furkawa, K. et al. 1997. The Actin-severing Protein Gelsolin Modulates Calcium Channel and NMDA Receptor Activities and Vulnerability to Excitotoxicity in Hippocampal Neurons. J. Neurosci 17: 8178-8186.

Gao, J. et al. 1994.  Transplacental Neurotoxic Effects of Monosodium Glutamate on Structures and Functions of Specific Brain Areas of Filial Mice. Sheng Li Hsueh Pao Acta Physiologica Sinica 46(1):44-51.

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Greene, James G. 1998. Bioenergeetics and Excitotoxicity: An Examination of Neurotoxicity Secondary to Succinate Dehydrogenase Inhibition. Ph.D. dissertation. The University of Rochester. Dissertation Abstracts Online. 59(03B): 0933.

Greenmayre, J. and Porter, R. 1994. Anatomy and Physiology of glutamate in the CNS. Neurol 44: s7-sl3.

Greenmayre, J. Timothy. 1998. Glutamatergic Mechanisms in the Cause and Treatment of Parkinson’s Disease. Speaker, NIH National Meeting. The Glutamate Cascade: Common Pathways of Central Nervous System Disease States. May3-5, 1998.

Hahn, J. S. et al. 1988. Central Mammalian Neurons Normally Resistant to Glutamate Toxicity Are Made Sensitive by Extracellular Ca Super (2+): Toxicity is blocked by the N-Methyl-D-Aspartate Antagonist.  Proceedings of the National Academy of Sciences USA 85 (17) 6556-6560.

Halpain, S., Hipolito, A. and Saffer, L. 1998. Regulation of F-actin Stability in Dendritic Spines by Glutamate Receptors and Calcineurin. J Neurosci 18: 9835-9844.

Helton, D.R. et al. 1998.  Anxiolytic and Side-Effect Profile of LY354740: A Potent, Highly Selective, Orally Active Agonist for Group II Metabotropic Glutamate Receptors. Journal of Pharmacology and Experimental Therapeutics 284(2):651-60.

Himi, T. et al. 1990. Spider Toxin (JSTX-3) Inhibits the Convulsions Induced by Glutamate Agonists. J. Neural Transm. 80 (2): 95-104.

Hussain, M.M. et al. 1989. Characterization of Glutamate Dehydrogenase Isoproteins Purified From the Cerebellum of Normal Subjects and Patients with Degenerative Neurological Disorders, and From Human Neoplastic Cell Lines. Journal of Biol. Chemistry 264 (34): 20730-735.

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Janssen, Raelyn et al. 1991. Glutamate Neurotoxicity in the Developing Rat Cochlea: Physiological and Morphological Approaches.   Brain Research 552 (2): 255-64.

Jensen, Frances E. 1998. Role of Glutamate Receptor Maturation in Perinatal Brain Injury. Speaker, NIH National Meeting. The Glutamate Cascade: Common Pathways of Central Nervous System Disease States. May3-5, 1998.

Kato, S. et al. 1990. Gliotoxic Effects of Alpha- Aminodipic Acid Isomers on the Carp Retina: A Long Term Observation. Neuroscience 36 (1): 145-153.

Kayastha, Arvind M. et al. 1991. Site-Directed Mutagenesis of the Beta Subunit of Tryptophan Synthase From Salmonella Typhimurium.   Role of Active Site Glutamic Acid. J. Biol. Chem. 266 (12): 7618-25.

Kirchgessner, A. L. et al. 1997.  Excitotoxicity in the enteric nervous system Journal of Neuroscience 17:8807-8816.

Kiskin, N. I. et al. 1989. Blocking Action of Nephalia Clavata Spider Toxin on Ionic Currents Activated by Glutamate and its Agonists in Isolated Hippocampal Neurons. Neurophysiology 21 (2): 110-116.

Klunk, W. E. et al. 1991. L Phosphoserine A Metabolite Elevated in Alzheimer's Disease Interacts with Specific L Glutamate Receptor Subtypes Journal of Neurochem 56 (6): 1997-2003.

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Kubo, T. et al. 1993.  Neonatal Glutamate Can Destroy the Hippocampal CA1 Structure and Impair Discrimination Learning in Rats Brain Research 616:311-314.

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Lipton, Stuart A. 1998. Excitotoxic Apoptosis and Necrosis in AIDS Dementia and Stroke: Potential Treatment with NMDA Open-channel Blockers and Nitric Oxide Related Species. Speaker, NIDA meeting, The Glutamate Cascade. Brigham and Women’s Hospital and Harvard Medical School.

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